34 research outputs found

    Multivariable linear regression models predicting serum TRAIL levels according to selected metabolic characteristics of study participants.

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    <p>Model 1 adjusted for age and gender.</p><p>Model 2 adjusted for age, gender and C-reactive protein.</p><p>Model 3 adjusted for age, gender, C-reactive protein and all variables included in table.</p><p>Model 4 Model 3 with stepwise backward selection of unnecessary variables (p for removal 0.1).</p

    Subcloning potential of EPC/ECFC generated from the PBMC of ACS patients.

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    <p>After <i>ex-vivo</i> expansion, primary EPC/ECFC colonies were trypsinized and assessed for clonogenic potential capacity by single cells replating assay. In <b>A</b>, single cells derived from EPC/ECPF colonies were seeded in collagen I coated wells and monitored day by day (<b>a</b>: day 1; <b>b</b>: day 2; <b>c</b>: day 3; <b>e–f</b>: day 4; <b>a–e</b>: original magnification 25X; <b>f</b>: original magnification 40X). One representative experiment is shown. In <b>B</b>, secondary clones were classified on the basis of their proliferation properties. Data are mean±SD derived from six independent experiments.</p

    Serum TRAIL levels in AMI patients and healthy individuals.

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    <p>Levels of TRAIL were determined by ELISA in sera from AMI patients (analyzed at the indicated times post AMI) and from healthy control subjects. Horizontal bars are median, upper and lower edges of box are 75th and 25th percentiles, lines extending from box are 10th and 90th percentiles.</p

    Serum TRAIL levels in AMI patient with respect to adverse clinical events.

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    <p>Serum levels of TRAIL were analyzed in AMI patient who died or experienced HF (with events) either during the acute phase (in-hospital) or in the follow-up (12 months after AMI) with respect to the other AMI patients (without events). Horizontal bars are median, upper and lower edges of box are 75th and 25th percentiles, lines extending from box are 10th and 90th percentiles.</p
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